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| Registros recuperados : 53 | |
| 4. |  | RUE, M. L. de la; HERRERA, H. M.; ALMEIDA, C. E.; SILVA, R. A. M. S. Alteracoes bioquimicas em caes durante infeccao aguda por Trypanosoma evansi (Steel, 1885) balbiani, 1888. In: CONGRESSO PANAMERICANO DE CIENCIAS VETERINARIAS, 15., 1996, Campo Grande. Abstracts... Campo Grande: SOMVET-MS / CRMV-MS, 1996. p.298| Biblioteca(s): Embrapa Pantanal. |
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| 7. | | LISBOA, C. V.; PINHO, A. P.; HERRERA, H. M.; GERHARDT, M.; CUPOLILLO, E.; JANSEN, A. M. Trypanosoma cruzi (kinetoplastida, Trypanosomatidae) genotypes in neotropical bats in Brazil. Veterinary Parasitology, v.156 , p.314-318, 2008.| Biblioteca(s): Embrapa Pantanal. |
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| 12. | | ALVES, F. M.; LIMA, J. S.; ROCHA, F. L.; HERRERA, H. M.; MOURAO, G.; JANSEN, A. M. Complexity and multi-factoriality of Trypanosoma cruzi sylvatic cycle in coatis, Nasua nasua (Procyonidae), and triatomine bugs in the Brazilian Pantanal. Parasites & Vectors, v. 9, n. 378, p. 1-9, 2016.| Biblioteca(s): Embrapa Pantanal. |
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| 14. | | RAMOS, V. do N.; PIOVEZAN, U.; FRANCO, A. H. A.; OSAVA, C. F.; HERRERA, H. M.; SZABÓ, M. P. J. Feral pigs as hosts for Amblyomma sculptum (Acari: Ixodidae) populations in the Pantanal, Mato Grosso do Sul, Brazil. Experimental & Applied Acarology, v. 64, n. 3, november 2014.| Biblioteca(s): Embrapa Pantanal. |
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| 16. | | HERRERA, H. M.; F. L. ROCHA.; LISBOA, C. V.; RADEMAKER, V.; MOURAO, G. de M.; JANSEN, A. M. Food web connections and the transmission cycles of Trypanosoma cruzi and Trypanosoma evansi (Kinetoplastida, Trypanosomatidae) in the PantanalRegion, Brazil. Transactions of the Royal Society of Tropical Medicine and Hygiene, v.7, n. 105, p. 380 -387, 2011.| Biblioteca(s): Embrapa Pantanal. |
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| 18. | | SILVA, R. A. M. S.; HERRERA, H. M.; DOMINGOS, L. B. da S.; XIMENES, F. A.; DAVILA, A. M. R. Pathogenesis of Trypanosoma evansi infection in dogs and horses: hematological and clinical aspects. Ciencia Rural, Santa Maria, v.25, n.2, p.233-238, maio/ago., 1995.| Biblioteca(s): Embrapa Pantanal. |
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 | Acesso ao texto completo restrito à biblioteca da Embrapa Pantanal. Para informações adicionais entre em contato com cpap.biblioteca@embrapa.br. |
Registro Completo
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Biblioteca(s): |
Embrapa Pantanal. |
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Data corrente: |
11/09/1997 |
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Data da última atualização: |
11/09/1997 |
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Autoria: |
SILVA, R. A. M. S.; RAMIREZ, L.; DAVILA, A. M. R.; PEREIRA, M. E. B.; HERRERA, H. M. |
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Afiliação: |
EMBRAPA. Centro de Pesquisa Agropecuaria do Pantanal (Corumba, MS); UFMS. Centro Universitario de Corumba (Corumba, MS). |
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Título: |
Infection in wistar rats after oral inoculation of Trypanosoma evansi. |
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Ano de publicação: |
1995 |
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Fonte/Imprenta: |
Memorias do Instituto Oswaldo Cruz, Rio de Janeiro, v.90, p.298, 1995. Suplemento I. |
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Idioma: |
Inglês |
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Conteúdo: |
Trypanosomosis due to Trypanosoma evansi represent one of the most important uncontrolled disease in the tropics and other parts of the world. These condition in specially prevalent in many developing countries and is a significant constrain to livestock production. T. evansi is the causative agent of "Mal de caderas" in the subtropical area of Argentina and Pantanal, Brazil. It is a salivarian trypanosome belonging to subgenus Trypanozoon and has no true intermediate hosts but it is transmitted directly or mechanically. In a similar manner that is observed in the stercorarian section, T. evansi demonstrated in this study a capacity to infect rats through oral route. Oral and intra-peritoneal (ip) inoculations of blood trypomastigotes as well as rats fed on infected carcasses resulted in 100% and 60% of rat infections respectivelly. The prepatent period of the parasitemia in animals inoculated orally with infected blood was eight days and nine days in rats fed on infected carcasses longer than those inoculated ip. All oral and i.p. infected animals died due to progressive infection. Three of five rats fed with infected carcasses also died. At necropsy, a striking feature was splenomegaly. The spleen was dark brown in colour, intensively congested and enlarged. The spleen of rats infected with blood enlarged twice from its normal size and eight times its normal weight. The spleen of rats fed on carcasses increased five times from its weight and aproximatelly two times its length. The spleen of rats inoculated i.p. increased two and half times its weight and only aproximatelly once and half its length. The liver and lungs were intensively hyperemic and the kidneys were congested in all infected animals. Similar alterations has been reported in T. brucei infection in mice and rats and by T. evansi in dogs. The high infectivity of T. evansi to rats through oral route could means that the oral important route in a sylvatic cycle. MenosTrypanosomosis due to Trypanosoma evansi represent one of the most important uncontrolled disease in the tropics and other parts of the world. These condition in specially prevalent in many developing countries and is a significant constrain to livestock production. T. evansi is the causative agent of "Mal de caderas" in the subtropical area of Argentina and Pantanal, Brazil. It is a salivarian trypanosome belonging to subgenus Trypanozoon and has no true intermediate hosts but it is transmitted directly or mechanically. In a similar manner that is observed in the stercorarian section, T. evansi demonstrated in this study a capacity to infect rats through oral route. Oral and intra-peritoneal (ip) inoculations of blood trypomastigotes as well as rats fed on infected carcasses resulted in 100% and 60% of rat infections respectivelly. The prepatent period of the parasitemia in animals inoculated orally with infected blood was eight days and nine days in rats fed on infected carcasses longer than those inoculated ip. All oral and i.p. infected animals died due to progressive infection. Three of five rats fed with infected carcasses also died. At necropsy, a striking feature was splenomegaly. The spleen was dark brown in colour, intensively congested and enlarged. The spleen of rats infected with blood enlarged twice from its normal size and eight times its normal weight. The spleen of rats fed on carcasses increased five times from its weight and aproximatelly two times its len... Mostrar Tudo |
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Thesaurus NAL: |
Pantanal; Trypanosoma evansi. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 02527naa a2200193 a 4500
001 1791636
005 1997-09-11
008 1995 bl uuuu u00u1 u #d
100 1 $aSILVA, R. A. M. S.
245 $aInfection in wistar rats after oral inoculation of Trypanosoma evansi.
260 $c1995
520 $aTrypanosomosis due to Trypanosoma evansi represent one of the most important uncontrolled disease in the tropics and other parts of the world. These condition in specially prevalent in many developing countries and is a significant constrain to livestock production. T. evansi is the causative agent of "Mal de caderas" in the subtropical area of Argentina and Pantanal, Brazil. It is a salivarian trypanosome belonging to subgenus Trypanozoon and has no true intermediate hosts but it is transmitted directly or mechanically. In a similar manner that is observed in the stercorarian section, T. evansi demonstrated in this study a capacity to infect rats through oral route. Oral and intra-peritoneal (ip) inoculations of blood trypomastigotes as well as rats fed on infected carcasses resulted in 100% and 60% of rat infections respectivelly. The prepatent period of the parasitemia in animals inoculated orally with infected blood was eight days and nine days in rats fed on infected carcasses longer than those inoculated ip. All oral and i.p. infected animals died due to progressive infection. Three of five rats fed with infected carcasses also died. At necropsy, a striking feature was splenomegaly. The spleen was dark brown in colour, intensively congested and enlarged. The spleen of rats infected with blood enlarged twice from its normal size and eight times its normal weight. The spleen of rats fed on carcasses increased five times from its weight and aproximatelly two times its length. The spleen of rats inoculated i.p. increased two and half times its weight and only aproximatelly once and half its length. The liver and lungs were intensively hyperemic and the kidneys were congested in all infected animals. Similar alterations has been reported in T. brucei infection in mice and rats and by T. evansi in dogs. The high infectivity of T. evansi to rats through oral route could means that the oral important route in a sylvatic cycle.
650 $aPantanal
650 $aTrypanosoma evansi
700 1 $aRAMIREZ, L.
700 1 $aDAVILA, A. M. R.
700 1 $aPEREIRA, M. E. B.
700 1 $aHERRERA, H. M.
773 $tMemorias do Instituto Oswaldo Cruz, Rio de Janeiro$gv.90, p.298, 1995. Suplemento I.
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